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Figure 3 Forearm blood flow at rest and during reactive hyperemia after the administration of L-NMMA, an NO synthase inhibitor, or indomethacin, a prostaglandin synthesis inhibitor, in hypertensive patients treated with ACE inhibitors (n = 13) or other agents (n = 15) and in untreated hypertensive patients (n = 18). L-NMMA abolished the enhancement of reactive hyperemia in hypertensive patients treated with ACE inhibitors (top). Indomethacin did not affect reactive hyperemia in any group (bottom).
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