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Influence of Pre-Existing Donor Atherosclerosis on the Development of Cardiac Allograft Vasculopathy and Outcomes in Heart Transplant Recipients

Haiyan Li, MD^_*, Koji Tanaka, MD, Hitoshi Anzai, MD, Brandy Oeser, MPH, Dominic Lai, BA, Jon A. Kobashigawa, MD and Jonathan M. Tobis, MD^,_*

^_* Department of Cardiology, Peking University Third Hospital, Beijing, China
Department of Medicine, Division of Cardiology, University of California Los Angeles, David Geffen School of Medicine, Los Angeles, California

View larger version (35K): [in a new window]   Figure 1 Bar graphs show that the cumulative cross-sectional area of stenosis was larger in donor lesions than in the two groups without donor lesions up to three years after transplantation (all p < 0.001). In sites without donor lesions, the cumulative cross-sectional area of stenosis was larger in arteries with donor lesions than in arteries without donor lesions up to three years after transplantation (all p < 0.01). The p value used one-way analysis of variance, followed by multiple comparisons with Bonferroni correction. DL = donor lesions; Without DL (1) = sites without donor lesions but from arteries with donor lesions; Without DL (2) = sites without donor lesions from arteries without donor lesions.

View larger version (13K): [in a new window]   Figure 2 Kaplan-Meier estimate of freedom from angiographic transplant coronary artery disease (TCAD) by three years after transplantation. Recipients with donor lesions: n = 89; recipients without donor lesions: n = 212.

View larger version (12K): [in a new window]   Figure 3 The cumulative cross-sectional area of stenosis at sites with and without donor lesions by three years after transplantation. DL = donor lesions; Sites Without DL (1) = sites without donor lesions but from arteries with donor lesions; Sites Without DL (2) = sites without donor lesions from arteries without donor lesions.