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J Am Coll Cardiol, 2008; 51:466-472, doi:10.1016/j.jacc.2007.08.060
© 2008 by the American College of Cardiology Foundation
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Relation Between Stress-Induced Myocardial Perfusion Defects on Cardiovascular Magnetic Resonance and Coronary Microvascular Dysfunction in Patients With Cardiac Syndrome X

Gaetano A. Lanza, MD*,*, Antonino Buffon, MD*, Alfonso Sestito, MD*, Luigi Natale, MD{dagger}, Gregory A. Sgueglia, MD*, Leda Galiuto, MD, FACC*, Fabio Infusino, MD*, Luca Mariani, MD*, Antonio Centola, MD* and Filippo Crea, MD, FACC*

* Istituto di Cardiologia, Università Cattolica del Sacro Cuore, Rome, Italy
{dagger} Istituto di Radiologia, Università Cattolica del Sacro Cuore, Rome, Italy.


Figure 1
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Figure 1 Coronary Flow Response to Adenosine in CSX Patients and Control Subjects

Individual and average (±SD) values of coronary blood flow response to adenosine in the left anterior descending (LAD) coronary artery, as assessed by transthoracic echo-Doppler, in cardiac syndrome X (CSX) patients and in healthy control subjects. The CSX patients are divided into those showing reversible perfusion defects (CSX LAD/CMR+) and those not showing any reversible regional perfusion defects (CSX LAD/CMR–) in the LAD territory on cardiovascular magnetic resonance (CMR) during dobutamine stress test.

 

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Figure 2 Myocardial Perfusion Defect on CMR During DST in a Patient With CSX

Cardiovascular perfusion magnetic resonance (CMR) first-pass study in a patient with cardiac syndrome X (CSX) (short-axis plane). In the left panels, obtained 26 s after administration of gadolinium at peak dobutamine stress test (DST), a perfusion defect is clearly visible in the mid-ventricular septum (arrows). The perfusion defect normalizes at rest (right).

 




 
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