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J Am Coll Cardiol, 2006; 48:1166-1174, doi:10.1016/j.jacc.2006.05.046 (Published online 25 August 2006).
© 2006 by the American College of Cardiology Foundation
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The T–786C Endothelial Nitric Oxide Synthase Genotype Predicts Cardiovascular Mortality in High-Risk Patients

Gian Paolo Rossi, MD*,*, Giuseppe Maiolino, MD*, Mario Zanchetta, MD{dagger}, Daniele Sticchi, PhD*, Luigi Pedon, MD{dagger}, Maurizio Cesari, MD*, Domenico Montemurro, MD*, Renzo De Toni, BsD*, Silvia Zavattiero, PhD* and Achille C. Pessina, MD, PhD*

* DMCS-Internal Medicine 4, University of Padua, Padua, Italy
{dagger} Servizio di Emodinamica and Divisione di Cardiologia Ospedale di Cittadella, University of Padua, Padua, Italy.


Figure 1
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  		Figure 1 The histograms showed the plasma levels of nitrate/nitrite (upper panel), nitrotyrosine (middle panel), and myeloperoxidase (bottom panel) in randomly selected CC (n = 88) and TT (n = 88) patients. The panels on the left showed all of the cohort; those on the right pertain to the CC (n = 26) and TT (n = 37) patients who were not receiving nitrate for treatment of coronary artery disease (CAD).

 

Figure 2
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  		Figure 2 Results of Kaplan-Meier analysis showing cardiovascular (CV) death-free survival in the high-risk patients divided by the T–786C endothelial nitric oxide synthase (eNOS) genotype. The TT homozygous patients had a significantly lower CV death-free survival.

 




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Copyright © 2006 by the American College of Cardiology Foundation.