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The T^–786C Endothelial Nitric Oxide Synthase Genotype Predicts Cardiovascular Mortality in High-Risk Patients

Gian Paolo Rossi, MD^_*,_*, Giuseppe Maiolino, MD^_*, Mario Zanchetta, MD, Daniele Sticchi, PhD^_*, Luigi Pedon, MD, Maurizio Cesari, MD^_*, Domenico Montemurro, MD^_*, Renzo De Toni, BsD^_*, Silvia Zavattiero, PhD^_* and Achille C. Pessina, MD, PhD^_*

^_* DMCS-Internal Medicine 4, University of Padua, Padua, Italy
Servizio di Emodinamica and Divisione di Cardiologia Ospedale di Cittadella, University of Padua, Padua, Italy.

View larger version (18K): [in a new window]   Figure 1 The histograms showed the plasma levels of nitrate/nitrite (upper panel), nitrotyrosine (middle panel), and myeloperoxidase (bottom panel) in randomly selected CC (n = 88) and TT (n = 88) patients. The panels on the left showed all of the cohort; those on the right pertain to the CC (n = 26) and TT (n = 37) patients who were not receiving nitrate for treatment of coronary artery disease (CAD).

View larger version (18K): [in a new window]   Figure 2 Results of Kaplan-Meier analysis showing cardiovascular (CV) death-free survival in the high-risk patients divided by the T–786C endothelial nitric oxide synthase (eNOS) genotype. The TT homozygous patients had a significantly lower CV death-free survival.