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The Influence of Percutaneous Closure of Patent Ductus Arteriosus on Left Ventricular Size and Function

A Prospective Study Using Two- and Three-Dimensional Echocardiography and Measurements of Serum Natriuretic Peptides

Department of Pediatric Cardiology, Hospital for Children and Adolescents, University of Helsinki, Helsinki, Finland

View larger version (24K): [in a new window]   Figure 1 Left ventricular (LV) diastolic volume adjusted to body surface area as measured by (A) two-dimensional (2D) echocardiography and (B) three-dimensional (3D) echocardiography at baseline, 1 day, and 6 months after patent ductus arteriosus (PDA) closure in the whole PDA group and in patients with normal-sized LV at baseline and in control group. *p < 0.05; p < 0.01; p < 0.001 as compared with control group; p < 0.05, ¶p < 0.01 within PDA group as compared with baseline.

View larger version (21K): [in a new window]   Figure 2 Serum concentrations of (A) N-terminal proatriopeptide (S-ANPN) and (B) N-terminal brain natriuretic peptide (S-proBNP) measured at baseline, 1 day, and 6 months after patent ductus arteriosus (PDA) closure in the whole PDA group and in patients with normal-sized left ventricle (LV) at baseline and in control subjects. *p < 0.05, p < 0.01, p < 0.001 as compared with control group; p < 0.05, ¶p < 0.01 within PDA group as compared with baseline.

View larger version (36K): [in a new window]   Figure 3 Aortic systolic, diastolic, and mean pressures measured in cardiac catheterization before and after patent ductus arteriosus (PDA) closure in the whole PDA group and in patients with normal-sized left ventricle (LV) at baseline. ¶p < 0.001 within the PDA group as compared with baseline.