AHA/ACCF Scientific Statement on the Evaluation of Syncope
From the American Heart Association Councils on Clinical Cardiology, Cardiovascular Nursing, Cardiovascular Disease in the Young, and Stroke, and the Quality of Care and Outcomes Research Interdisciplinary Working Group; and the American College of Cardiology Foundation In Collaboration With the Heart Rhythm Society
S. Adam Strickberger, MD,
D. Woodrow Benson, MD, PhD,
Italo Biaggioni, MD,
David J. Callans, MD,
Mitchell I. Cohen, MD,
Kenneth A. Ellenbogen, MD,
Andrew E. Epstein, MD,
Paul Friedman, MD,
Jeffrey Goldberger, MD,
Paul A. Heidenreich, MD,
George J. Klein, MD,
Bradley P. Knight, MD,
Carlos A. Morillo, MD,
Robert J. Myerburg, MD and
Cathy A. Sila, MD, FAHA
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Figure 1 Flow chart for the diagnostic approach to the patient with syncope.
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Figure 2 Different patterns of QT prolongation in LQTS. Morphology of the QT segment and T wave may be different in different genetic subsets of the LQTS, although there is significant individual variation. Reproduced with permission from Moss et al.(64)
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Figure 3 ECG changes in the Brugada syndrome. ST elevation occurs in the anterior precordial leads, leads V1 and V2. Type 1 (coved) ECGs with  1 mV of ST elevation have the most prognostic significance. ECG recordings may change over time, as in this example, and serial ECGs may be important. Reproduced with permission from Wilde et al.(65)
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