Advertisement






Click here for more guidelines.
CME Topic Collections Past Issues Search Current Issue Home
     

J Am Coll Cardiol, 2006; 47:2470-2476, doi:10.1016/j.jacc.2006.01.072 (Published online 24 May 2006).
© 2006 by the American College of Cardiology Foundation
This Article
Right arrow Abstract Freely available
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Web of Science (6)
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Li, H.
Right arrow Articles by Tobis, J. M.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Li, H.
Right arrow Articles by Tobis, J. M.

Influence of Pre-Existing Donor Atherosclerosis on the Development of Cardiac Allograft Vasculopathy and Outcomes in Heart Transplant Recipients

Haiyan Li, MD*, Koji Tanaka, MD{dagger}, Hitoshi Anzai, MD{dagger}, Brandy Oeser, MPH{dagger}, Dominic Lai, BA{dagger}, Jon A. Kobashigawa, MD{dagger} and Jonathan M. Tobis, MD{dagger},*

* Department of Cardiology, Peking University Third Hospital, Beijing, China
{dagger} Department of Medicine, Division of Cardiology, University of California Los Angeles, David Geffen School of Medicine, Los Angeles, California.


Figure 1
View larger version (42K):

[in a new window]
 
Figure 1 Bar graphs show that the cumulative cross-sectional area of stenosis was larger in donor lesions than in the two groups without donor lesions up to three years after transplantation (all p < 0.001). In sites without donor lesions, the cumulative cross-sectional area of stenosis was larger in arteries with donor lesions than in arteries without donor lesions up to three years after transplantation (all p < 0.01). The p value used one-way analysis of variance, followed by multiple comparisons with Bonferroni correction. DL = donor lesions; Without DL (1) = sites without donor lesions but from arteries with donor lesions; Without DL (2) = sites without donor lesions from arteries without donor lesions.

 

Figure 2
View larger version (22K):

[in a new window]
 
Figure 2 Kaplan-Meier estimate of freedom from angiographic transplant coronary artery disease (TCAD) by three years after transplantation. Recipients with donor lesions: n = 89; recipients without donor lesions: n = 212.

 

Figure 3
View larger version (20K):

[in a new window]
 
Figure 3 The cumulative cross-sectional area of stenosis at sites with and without donor lesions by three years after transplantation. DL = donor lesions; Sites Without DL (1) = sites without donor lesions but from arteries with donor lesions; Sites Without DL (2) = sites without donor lesions from arteries without donor lesions.

 




 
  CME Topic Collections Past Issues Search Current Issue Home

Advertisement