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Aspirin Resistance and Atherothrombotic Disease

Whitaker Cardiovascular Institute and Evans Department of Medicine, Boston University School of Medicine, Boston, Massachusetts.

View larger version (33K): [in a new window]   Figure 1 Platelet function and mechanisms of antiplatelet therapy. ADP = adenosine diphosphate; Ecs = endothelial cells; Gi = inhibitory G protein; GP = glycoprotein; PG = prostaglandin; P2 = type 2 platelet purinergic receptor; TX = thromboxane; HETE = hydroxyeicosatetraenoic acid; HPETE = hydroperoxyeicosatetraenoic acid.