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Cardiovascular morbidity and mortality in hypertensive patients with a history of atrial fibrillation

The Losartan Intervention For End point reduction in hypertension (LIFE) study

Kristian Wachtell, MD, PhD^*,,*, Björn Hornestam, MD, PhD, Mika Lehto, MD, David J. Slotwiner, MD, FACC, Eva Gerdts, MD, PhD^||, Michael H. Olsen, MD, PhD^*, Peter Aurup, MD^¶, Björn Dahlöf, MD, PhD, FACC, Hans Ibsen, MD^*, Stevo Julius, MD, FACC^#, Sverre E. Kjeldsen, MD, PhD, FACC^**, Lars H. Lindholm, MD, Markku S. Nieminen, MD, Jens Rokkedal, MD^* and Richard B. Devereux, MD, FACC

^* Department of Medicine, Glostrup University Hospital, Glostrup, Denmark
Division of Cardiology, Weill Medical College of Cornell University, New York, New York
Department of Medicine, Sahlgrenska University Hospital/Östra, Göteborg, Sweden
Department of Cardiology, Helsinki University Central Hospital, Helsinki, Finland
^|| Department of Cardiology, Haukeland University Hospital, Bergen, Norway
^¶ Merck Research Laboratories, West Point, Pennsylvania
^# Department of Medicine, University of Michigan, Ann Arbor, Michigan
^** Department of Preventive Medicine, Umeå University Hospital, Umeå, Sweden
Department of Medicine, Ullevål University Hospital, Oslo, Norway

View larger version (15K): [in a new window]   Figure 1 Systolic, diastolic, and mean arterial blood pressure in hypertensive patients with atrial fibrillation during follow-up; intention-to-treat analysis.

View larger version (11K): [in a new window]   Figure 2 Heart rate in hypertensive patients with a history of atrial fibrillation treated with losartan and atenolol during follow-up. *p < 0.01. **p < 0.001.

View larger version (28K): [in a new window]   Figure 3 Kaplan-Meier curves in each treatment group of hypertensive patients with a history of atrial fibrillation for occurrence of (A) the primary composite end point; (B) cardiovascular death; (C) fatal or non-fatal myocardial infarction; (D) fatal or non-fatal stroke; and (E) all-cause mortality.