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J Am Coll Cardiol, 2004; 44:1700-1706, doi:10.1016/j.jacc.2004.08.034
© 2004 by the American College of Cardiology Foundation
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Mapping and ablation of polymorphic ventricular tachycardia after myocardial infarction

Lukasz Szumowski, MD, PhD*,*, Prashanthan Sanders, MBBS, PhD{dagger}, Franciszek Walczak, MD, PhD*, Mélèze Hocini, MD{dagger}, Pierre Jaïs, MD{dagger}, Roman Kepski, PhD*, Ewa Szufladowicz, MD, PhD*, Piotr Urbanek, MD*, Pawel Derejko, MD*, Robert Bodalski, MD* and Michel Haïssaguerre, MD{dagger}

* Institute of Cardiology, Warsaw, Poland
{dagger} Hopital Cardiologique du Haut-Leveque and the Université Victor Segalen Bordeaux II, Bordeaux, France



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Figure 1 Examples of frequent isolated premature beats followed by the initiation of polymorphic ventricular tachycardia.

 


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Figure 2 Electroanatomic bipolar voltage map of Patient #4 performed during sinus rhythm. The voltage map delineates the region of scar border-zone. Note that the premature beats originating from the Purkinje network are in this border-zone, where successful ablation was performed (brown tags). Purple tags represent sites recording Purkinje potentials in sinus rhythm, and white tags represent fractionated complex electrograms.

 


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Figure 3 Electroanatomic bipolar voltage and activation map in Patient #3 performed during premature beats. The activation map demonstrates the centrifugal activation from the border-zone of the scar. The voltage map (right panel) delineates the region of scar border-zone. Note that the premature beats originating from the Purkinje network are in this border-zone, where successful ablation was performed (brown tags). Purple tags represents sites recording Purkinje potentials, and white tags represent fractionated complex electrograms.

 


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Figure 4 Mapping at the site of earliest activity during premature beats. Note that the Purkinje potential (*) precedes the ventricular activation during both sinus rhythm and the premature beat. Also, the width of the premature beat is quite similar to that during sinus rhythm.

 


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Figure 5 Purkinje potential (*) preceding each beat of tachycardia, suggestive of driving the tachycardia. The morphology of the ventricular complexes, originating from the Purkinje fibers, is changing, suggesting different Purkinje-muscle outbreak.

 


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Figure 6 Purkinje potential preceding both sinus and premature beats (*). The last beat demonstrates Purkinje to muscle conduction block.

 




 
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