Hemodynamic effects of inhaled nitric oxide in right ventricular myocardial infarction and cardiogenic shock
Ignacio Inglessis, MD*,
Jordan T. Shin, MD*, ,
John J. Lepore, MD*,
Igor F. Palacios, MD, FACC*,
Warren M. Zapol, MD ,
Kenneth D. Bloch, MD*, and
Marc J. Semigran, MD*,*
* Cardiology Division
Cardiovascular Research Center
Department of Anesthesia and Critical Care, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts, USA

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Figure 1 Hemodynamic parameters in right ventricular myocardial infarction patients before (Pre-nitric oxide [NO]), during (NO), and 10 min after the discontinuation of NO inhalation (Post-NO). RAP = mean right atrial pressure; PAP = mean pulmonary arterial pressure; PVR = pulmonary vascular resistance; CI = cardiac index; PCWP = mean pulmonary capillary wedge pressure; SAP = mean systemic arterial pressure; SVR = systemic vascular resistance; HR = heart rate. *p < 0.05 versus Pre-NO and Post-NO; p < 0.05 versus Pre-NO.
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Figure 2 Right-to-left shunt flow and systemic arterial oxygen saturation in three right ventricular myocardial infarction patients before (Pre-nitric oxide [NO]), during (NO), and 10 min after the discontinuation of NO inhalation (Post-NO). Diamonds = Patient #1; squares = Patient #5; triangles = Patient #4.
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