Effects of prior moderate exercise on postprandial metabolism and vascular function in lean and centrally obese men
Jason M.R. Gill, PhD*,*,
Ali Al-Mamari, MD ,
William R. Ferrell, MD, PhD,
Stephen J. Cleland, MD, PhD ,
Chris J. Packard, DSc*,
Naveed Sattar, MD, PhD*,
John R. Petrie, MD, PhD and
Muriel J. Caslake, PhD*
* Departments of Vascular Biochemistry
Medicine, University of Glasgow, Glasgow Royal Infirmary, Glasgow, United Kingdom
Department of Medicine and Therapeutics, University of Glasgow, Western Infirmary, Glasgow, United Kingdom
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Figure 1 Postprandial plasma triglyceride (TG) concentrations (top panels), insulin (second row of panels), glucose (third row of panels), and nonesterified fatty acid (NEFA) (bottom panels) concentrations in the lean (n =10, left panels) and centrally obese (n = 10, right panels) in the control and exercise trials. Summary statistics for these responses are shown in Table 3.
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Figure 2 Microvascular responses to acetylcholine (Ach) (top panel) and sodium nitroprusside (SNP) (bottom panel) in the fasted and postprandial states for the lean and centrally obese subjects combined in the control and exercise trials (n = 20). Statistics performed on logarithmically transformed data. *Different from fasting value in the same trial, p < 0.05 (**p < 0.01); difference between control and exercise trials at the same time point, p < 0.05.
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Figure 3 Postprandial plasma interleukin-6 (IL-6) (top panels) and white blood cell counts (bottom panels) in the lean (n =10, left panels) and centrally obese (n = 10, right panels) in the control and exercise trials. *Different from fasting value in the same trial, p < 0.05 (**p < 0.01); difference between control and exercise trials at the same time point, p < 0.05.
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