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Wall stress modulates brain natriuretic peptide production in pressure overload cardiomyopathy

Marc Vanderheyden, MD^*,*, Marc Goethals, MD^*, Sofie Verstreken, MD^*, Bernard De Bruyne, MD^*, Kristin Muller, RN, Eddy Van Schuerbeeck, RN^* and Jozef Bartunek, MD^*

^* Cardiovascular Center, Onze Lieve Vrouw Ziekenhuis, Aalst, Belgium
Cardiovascular Center, Imelda Ziekenhuis, Bonheiden, Belgium

View larger version (16K): [in a new window]   Figure 1 Serum B-type natriuretic peptide (BNP) levels in age-matched controls (NI), aortic stenosis patients with normal (group A) and with impaired preload reserve (group B).

View larger version (11K): [in a new window]   Figure 2 Correlations between log-transformed (Ln) B-type natriuretic peptide (BNP) and parameters of contractile function. FS = fractional shortening; LV = left ventricular; PCWP = pulmonary capillary wedge pressure.

View larger version (13K): [in a new window]   Figure 3 Correlations between log-transformed (Ln) B-type natriuretic peptide (BNP) and parameters of diastolic load. IVRT = isovolumic relaxation time; LVEDVI = indexed left ventricular end-diastolic volume.