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Impaired intravascular triglyceride lipolysis constitutes a marker of clinical outcome in patients with stable angina undergoing secondary prevention treatment

A long-term follow-up study

Andrei C. Sposito, MD, PhD^*, Pedro A. Lemos, MD, Raul D. Santos, MD, PhD, Whady Hueb, MD, PhD, Carmen G. C. Vinagre, PhD, Edgard Quintella, MD, Otavio Carneiro, MD, M. John Chapman, PhD, DSc, Jose A. F. Ramires, MD, PhD, FACC^* and Raul C. Maranhão, MD, PhD^,*

^* Heart Institute (InCor), Zerbini Foundation, Brasilia, Brazil
Heart Institute (InCor), University of São Paulo Medical School, São Paulo, Brazil
INSERM Unité 551, Hôpital de la Pitié-Salpetriere, Paris, France
Faculty of Pharmaceutical Sciences, University of São Paulo, São Paulo, Brazil

View larger version (18K): [in a new window]   Figure 1 Plasma disappearance curves of the emulsion 3H-triglyceride (TG) (A) and 14C-cholesteryl ester (CE) (B) determined in the controls, in patients who presented clinical aggravating coronary artery disease (ACAD), and in those who had evolution of stable coronary artery disease (SCAD).

View larger version (20K): [in a new window]   Figure 2 Kaplan-Meier curves of the time elapsed to a refractory angina event in the two groups of patients with delipidation index (DI) above or below the median. CI = confidence interval.