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Angiographic variables predict increased riskfor adverse ischemic events after coronarystenting with glycoprotein IIb/IIIa inhibition

Results from the TARGET trial

Mitchell J. Ross, MD^*, Howard C. Herrmann, MD, FACC^*,*, David J. Moliterno, MD, FACC, James C. Blankenship, MD, FACC, Laura Demopoulos, MD, Peter M. DiBattiste, MD, FACC, Stephen G. Ellis, MD, FACC, Ziyad Ghazzal, MD, FACC^||, Jack L. Martin, MD, FACC^¶, Jennifer White and Eric J. Topol, MD, FACC

^* Division of Cardiology, University of Pennsylvania Medical Center, Philadelphia, Pennsylvania, USA
Division of Cardiology, Cleveland Clinic Foundation, Cleveland, Ohio, USA
Division of Cardiology, Geisinger Medical Center, Danville, Pennsylvania, USA
Merck & Co., Inc., West Point, Pennsylvania, USA
^|| Division of Cardiology, Emory University School of Medicine, Atlanta, Georgia, USA
^¶ Division of Cardiology, Bryn Mawr Hospital, Bryn Mawr, Pennsylvania, USA

View larger version (25K): [in a new window]   Figure 1 Incidence of 30-day (A) and 6-month (B) primary composite end point according to the presence of high-risk angiographic features. *p < 0.05. MI = myocardial infarction; TVR = target vessel revascularization.

View larger version (18K): [in a new window]   Figure 2 Incidence of the 30-day primary composite end point according to the presence of angiographic variables: thrombus, lesion eccentricity, or lesion length >20 mm. MI = myocardial infarction; TVR = target vessel revascularization.

View larger version (19K): [in a new window]   Figure 3 Incidence of the six-month secondary composite end point according to the presence of angiographic variables: thrombus, lesion eccentricity, or lesion length >20 mm. MI = myocardial infarction; TVR = target vessel revascularization.