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Do associations with C-Reactive protein and extent of coronary artery disease account for the increased cardiovascular risk of renal insufficiency?

James S. Zebrack, MD, Jeffrey L. Anderson, MD, FACC^*,*, Srinivasan Beddhu, MD, Benjamin D. Horne, MStat, MPH^*, Tami L. Bair, BS^*, Alfred Cheung, MD, Joseph B. Muhlestein, MD, FACC^* Intermountain Heart Collaborative Study Group

^* Cardiovascular Department, LDS Hospital, Salt Lake City, Utah, USA
Divisions of Cardiology and Nephrology, University of Utah School of Medicine, Salt Lake City, Utah, USA

View larger version (19K): [in a new window]   Figure 1 Kaplan-Meier event-free (first event of death or myocardial infarction [MI]) survival curves for patients with glomerular filtration rate GFR <60 mg/dl (+renal insufficiency [RI], n = 77), C-reactive protein (CRP) >1.0 mg/dl (+CRP, n = 769), both (n = 303) or neither (n = 335).

View larger version (17K): [in a new window]   Figure 2 Absolute percentage of death or myocardial infarction (MI) among patients with glomerular filtration rate <60 mg/dl (+ renal insufficiency [RI]), C-reactive protein (CRP) >1.0 mg/dl (+CRP), both or neither, during mean follow-up of three years. Solid bars = MI; open bars = death.