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Troponin and C-reactive protein have different relations to subsequent mortality and myocardial infarction after acute coronary syndrome

A GUSTO-IV substudy

Stefan K. James, MD^*,*, Paul Armstrong, MD, Elliott Barnathan, MD, PhD, Robert Califf, MD, Bertil Lindahl, MD, PhD^*, Agneta Siegbahn, MD, PhD^||, Maarten L. Simoons, MD, PhD^¶, Eric J. Topol, MD^#, Per Venge, MD, PhD^||, Lars Wallentin, MD, PhD^* GUSTO-IV–ACS Investigators

^* Department of Medical Sciences, Cardiology, Uppsala, Sweden
^|| Clinical Chemistry, Uppsala, Sweden
Department of Medicine, Alberta, Canada
Centocor, Malvern, Pennsylvania, USA
Duke CRI, Durham, North Carolina, USA
^¶ Thoraxcenter, Erasmus Medical Center, Rotterdam, The Netherlands
^# Cleveland Clinic Foundation, Cleveland, Ohio, USA

View larger version (32K): [in a new window]   Figure 1 Rate of (a) death/myocardial infarction (MI), (b) death, and (c) MI at 48 h, 7 days, and 30 days, respectively, in relation to quartiles of troponin T. The number of patients with events is noted under the bars.

View larger version (33K): [in a new window]   Figure 2 Rate of (a) death/myocardial infarction (MI), (b) death, and (c) MI at 48 h, 7 days, and 30 days, respectively, in relation to quartiles of C-reactive protein (CRP). The number of patients with events is noted under the bars.