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Association of albuminuria with systolic and diastolic left ventricular dysfunction in type 2 diabetes

The Strong Heart Study

Jennifer E. Liu, MD, FACC^,*, David C. Robbins, MD, Vittorio Palmieri, MD^*, Jonathan N. Bella, MD, FACC^*, Mary J. Roman, MD, FACC^*, Richard Fabsitz, MA, Barbara V. Howard, PhD, Thomas K. Welty, MD, MPH^||, Elisa T. Lee, PhD^¶ and Richard B. Devereux, MD, FACC^*

^* Department of Medicine, the New York Hospital-Cornell Medical Center, New York, New York, USA
Division of Cardiology, the New York Hospital-Cornell Medical Center, New York, New York, USA
National Heart, Lung, and Blood Institute, Bethesda, Maryland, USA
Medstar Research Institute, Washington, DC, USA
^|| Aberdeen Area Tribal Chairmen’s Health Board, Rapid City, South Dakota, USA
^¶ School of Public Health, University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma, USA

View larger version (19K): [in a new window]   Figure 1 Prevalence of subnormal midwall shortening (MWS) based on albuminuria status, where I = no albuminuria; II = microalbuminuria; and III = macroalbuminuria. Solid bars = percentage of subnormal MWS. Comparison between each group was made using chi-squared statistics with Bonferroni correction.

View larger version (22K): [in a new window]   Figure 2 Prevalence of abnormal diastolic function based on albuminuria status where I = no albuminuria; II = microalbuminuria; and III = macroalbuminuria. Solid bars = percentage of abnormal diastolic function. Comparison between each group was made using chi-squared statistics with Bonferroni correction.