Sympathetic neural mechanisms in white-coat hypertension
Paul A. Smith, MB, ChB*,*,
Lee N. Graham, MB, ChB*,
Alan F. Mackintosh, MA, MD*,
John B. Stoker, BSc, MB, ChB* and
David A. S. G. Mary, MB, ChB, PhD*
* Department of Cardiology, Leeds Teaching Hospitals, Leeds, United Kingdom

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Figure 1 The mean frequency of single-unit muscle sympathetic nerve activity (s-MSNA) in the three groups of subjects, NT (normotension), WHT (white-coat hypertension) and EHT (essential hypertension) expressed as mean (height of columns) and SEM (bars). Symbols represent statistical analysis between groups; *p < 0.05 and p < 0.05.
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Figure 2 The mean frequency of muscle sympathetic nerve activity (MSNA) bursts in the three groups of subjects, NT (normotension), WHT (white coat hypertension) and EHT (essential hypertension) expressed as mean (height of columns) and SEM (bars). Symbols represent statistical analysis between groups; *p < 0.05 and p < 0.05.
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Figure 3 Cardiac baroreceptor sensitivity (BRS) in the three groups of subjects, NT (normotension), WHT (white coat hypertension) and EHT (essential hypertension) expressed as mean (height of columns) and SEM (bars). Symbols represent statistical analysis between groups; *p > 0.05 and p < 0.05.
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