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J Am Coll Cardiol, 2002; 39:1456-1463
© 2002 by the American College of Cardiology Foundation
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Revisiting the culprit lesion in non–Q-wave myocardial infarction

Results from the VANQWISH trial angiographic core laboratory

Richard A. Kerensky, MD*,*, Michael Wade, MS{dagger}, Prakash Deedwania, MD{ddagger}, William E. Boden, MD{dagger}§, Carl J. Pepine, MD, MACC* Veterans Affairs Non–Q-Wave Infarction Strategies in-Hospital (VANQWISH) Trial Investigators

* Malcom Randall Veterans Affairs Medical Center and the University of Florida College of Medicine, Division of Cardiovascular Medicine, Gainesville, Florida, USA
{dagger} Veterans Affairs Medical Center and the State University of New York Health Science Center, Syracuse, New York, USA
{ddagger} Veterans Affairs Medical Center, Fresno, California, USA
§ Division of Cardiology at Hartford Hospital, Hartford, Connecticut, USA



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Figure 1 Rates of revascularization with percutaneous transluminal coronary angioplasty (PTCA) or coronary artery bypass grafting (CABG) in patients with no culprit lesion, one culprit lesion or multiple culprit lesions. Patients with one culprit lesion are divided into two groups: incomplete occlusion or acute total occlusion. *Single incomplete occlusion vs. no or multiple culprit lesions, p < 0.001; **Single incomplete occlusion vs. single acute occlusion, p = 0.128; ***Multiple culprit lesions vs. other groups, p = 0.010.

 




 
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