Radial, carotid and aortic distensibility in congestive heart failure: effects of high-dose angiotensin-converting enzyme inhibitor or low-dose association with angiotensin type 1 receptor blockade
Cristina Giannattasio, MD, PhD* ,
Felice Achilli, MD ,
Monica Failla, MD*,
Anna Capra, MD*,
Antonella Vincenzi, MD,
Franco Valagussa, MD and
Giuseppe Mancia, MD*,*
* Clinica Medica, Department of Medicina Clinica, Prevenzione e Biotecnologie Sanitarie, Milano-Bicocca University, Milano, Italy
Divisione di Cardiologia, S. Gerardo Hospital, Monza, Italy
IRCCS Istituto Auxologico, Milano, Italy
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Figure 1 Radial artery (RA) and carotid artery (CA) wall thickness and RA, CA and abdominal aorta distensibility in control subjects (n = 30) and in patients with congestive heart failure (n = 30). Data are shown as mean ± SE. Open bar = controls (n = 30); hatched bar = heart failure (n = 30).
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Figure 2 Radial artery (RA) distensibility-pressure curves in the control and congestive heart failure groups of Figure 1. Open circle = controls, n = 30; open square = heart failure, n = 30.
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Figure 3 Radial artery (RA), carotid artery (CA) and abdominal aorta (AO) distensibility in 30 patients with congestive heart failure taking three different treatment regimens at baseline and two months later. Data are shown as mean ± SE. Open bar = control; hatched bar = treatment, two months. ACEI = angiotensin-converting enzyme inhibitor; ATA = angiotensin II antagonist; T = treatment.
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