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Deconditioning fails to explain peripheral skeletal muscle alterations in men with chronic heart failure

Brian D. Duscha, MS^*,*, Brian H. Annex, MD^*,, Howard J. Green, PhD, Anne M. Pippen, MS^* and William E. Kraus, MD

^* Department of Medicine, Division of Cardiology, Duke University Medical Center, Durham, North Carolina, USA
Department of Cell Biology, Duke University Medical Center, Durham, North Carolina, USA
Department of Medicine, Division of Cardiology, Durham Veterans Administration Medical Center, Durham, North Carolina, USA
Department of Kinesiology, University of Waterloo, Waterloo, Ontario, Canada

View larger version (16K): [in a new window]   Figure 1 Comparison of capillary density (endothelial cells/muscle fiber; mean ± SE) between men and women with chronic heart failure (CHF) and normal subjects (*p < 0.02).

View larger version (19K): [in a new window]   Figure 2 Comparison of the oxidative enzyme 3-hydroxyl coenzyme A (CoA) dehydrogenase (3-HAD) activity (mol/kg/protein; mean ± SE) between men and women with chronic heart failure (CHF) and normal subjects (*p = 0.02).