Impact of normalized myocardial perfusion after successful angioplasty in acute myocardial infarction
Gregg W. Stone, MD, FACC*,*,
Michael A. Peterson, MD ,
Alexandra J. Lansky, MD, FACC*,
George Dangas, MD, FACC*,
Roxana Mehran, MD, FACC* and
Martin B. Leon, MD, FACC*
* Cardiovascular Research Foundation, Lenox Hill Heart and Vascular Institute, New York, New York, USA
The Washington Hospital Center, Washington, DC, USA
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Figure 1 Rates of Thrombolysis In Myocardial Infarction (TIMI) flow grades and myocardial blush scores by separate core laboratory analysis before (left) and after (right) procedure.
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Figure 2 Infarct size as estimated by peak serum creatine phosphokinase-MB isozyme (CPK-MB) levels after intervention, stratified by myocardial blush score, in all 173 patients (left) and in 163 patients in whom Thrombolysis In Myocardial Infarction (TIMI)-3 flow was also restored (right).
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Figure 3 Cumulative survival (including in-hospital and late events) after percutaneous intervention in 173 patients with acute myocardial infarction, stratified by final procedural Thrombolysis In Myocardial Infarction (TIMI) flow (left) and myocardial blush score (right). P values represent log rank for trend.
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Figure 4 Cumulative survival after intervention in 163 patients in whom Thrombolysis In Myocardial Infarction (TIMI)-3 flow was achieved, stratified by status of myocardial perfusion.
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Figure 5 Cumulative survival after intervention in 163 patients in whom Thrombolysis In Myocardial Infarction (TIMI)-3 flow was achieved, stratified by status of myocardial perfusion, and subgrouped by myocardial infarct distribution (left) and the presence or absence of cardiogenic shock at baseline (right).
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