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Impact of normalized myocardial perfusion after successful angioplasty in acute myocardial infarction

Gregg W. Stone, MD, FACC^*,*, Michael A. Peterson, MD, Alexandra J. Lansky, MD, FACC^*, George Dangas, MD, FACC^*, Roxana Mehran, MD, FACC^* and Martin B. Leon, MD, FACC^*

^* Cardiovascular Research Foundation, Lenox Hill Heart and Vascular Institute, New York, New York, USA
The Washington Hospital Center, Washington, DC, USA

View larger version (19K): [in a new window]   Figure 1 Rates of Thrombolysis In Myocardial Infarction (TIMI) flow grades and myocardial blush scores by separate core laboratory analysis before (left) and after (right) procedure.

View larger version (31K): [in a new window]   Figure 2 Infarct size as estimated by peak serum creatine phosphokinase-MB isozyme (CPK-MB) levels after intervention, stratified by myocardial blush score, in all 173 patients (left) and in 163 patients in whom Thrombolysis In Myocardial Infarction (TIMI)-3 flow was also restored (right).

View larger version (18K): [in a new window]   Figure 3 Cumulative survival (including in-hospital and late events) after percutaneous intervention in 173 patients with acute myocardial infarction, stratified by final procedural Thrombolysis In Myocardial Infarction (TIMI) flow (left) and myocardial blush score (right). P values represent log rank for trend.

View larger version (28K): [in a new window]   Figure 4 Cumulative survival after intervention in 163 patients in whom Thrombolysis In Myocardial Infarction (TIMI)-3 flow was achieved, stratified by status of myocardial perfusion.

View larger version (18K): [in a new window]   Figure 5 Cumulative survival after intervention in 163 patients in whom Thrombolysis In Myocardial Infarction (TIMI)-3 flow was achieved, stratified by status of myocardial perfusion, and subgrouped by myocardial infarct distribution (left) and the presence or absence of cardiogenic shock at baseline (right).