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An inhibitor of inducible nitric oxide synthase decreases forearm blood flow in patients with congestive heart failure

Yutaka Ishibashi, MD^*,*, Toshio Shimada, MD^*, Yo Murakami, MD^*, Nobuyuki Takahashi, MD^*, Takeshi Sakane, MD^*, Takashi Sugamori, MD^*, Shuzo Ohata, MD^*, Shin-ichi Inoue, MD^*, Yoko Ohta, MD^*, Ko Nakamura, MD^*, Hiromi Shimizu, MD^*, Harumi Katoh, MD^* and Michio Hashimoto, MD

^* Fourth Department of Internal Medicine, Izumo, Japan
Department of Physiology, Shimane Medical University, Izumo, Japan

View larger version (21K): [in a new window]   Figure 1 Percent change from baseline in the forearm blood flow (FBF) ratio between the infused and noninfused arms during intra-arterial infusion of aminoguanidine (left) and NG-monomethyl-L-arginine (L-NMMA) (right) in individual patients with congestive heart failure.

View larger version (19K): [in a new window]   Figure 2 Time course of forearm blood flow (FBF) after finishing infusion of the last dose of aminoguanidine in 13 patients with congestive heart failure. Data are presented as the mean value ± SD.

View larger version (21K): [in a new window]   Figure 3 Percent change from baseline in the forearm blood flow (FBF) ratio between the infused and noninfused arms during intra-arterial infusion of acetylcholine under co-infusion of saline, aminoguanidine or NG-monomethyl-L-arginine (L-NMMA) in five normal subjects. *p < 0.001 vs. corresponding saline value.