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Evidence supporting abnormalities in nitric oxide synthase function induced by nitroglycerin in humans

Tommaso Gori, MD^* , Susanna S. Mak, MD^*, Susan Kelly, RN^* and John D. Parker, MD, FACC^*

^* Division of Cardiology, Department of Medicine, Mount Sinai Hospital, Toronto, Canada
Cardiovascular Collaborative Program, University of Toronto, Toronto, Canada

View larger version (10K): [in a new window]   Figure 1 Responses to acetylcholine (Ach) infusion (expressed as change in ratio of infused to noninfused arm). Open bar = control group; solid bar = GTN group. FBF = forearm blood flow; GTN = nitroglycerin; I/N = ratio of forearm blood flow, infused to noninfused arm.

View larger version (10K): [in a new window]   Figure 2 Responses to N-monomethyl-L-arginine (L-NMMA) infusion (expressed as change in ratio of infused to noninfused arm). Open bar = control group; solid bar = GTN group. Abbreviations as in Figure 1.

View larger version (11K): [in a new window]   Figure 3 Responses to sodium nitroprusside infusion (expressed as change in ratio of infused to noninfused arm). Open bar = control group; solid bar = GTN group. SNP = sodium nitroprusside. Other abbreviations as in Figure 1.