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Pressure-guided nonsurgical myocardial reduction induced by small septal infarctions in hypertrophic obstructive cardiomyopathy

^a Internal Medicine I, Klinikum Grosshadern, Ludwig-Maximilians-University, Munich, Germany

View larger version (18K): [in a new window]   Figure 1 Invasively determined left ventricular outflow tract (LVOT) gradient before, immediately after, four to six months after and 12 to 18 months after nonsurgical myocardial reduction. *p < 0.001 compared with baseline. n.s. = statistically nonsignificant.

View larger version (22K): [in a new window]   Figure 2 Development of left ventricular hypertrophy after nonsurgical myocardial reduction. Septal segments = volume of eight septal segments; contralateral segments = volume of eight segments of the lateral wall; total segments = volume of 64 segments. *p < 0.001 compared with baseline. n.s. = statistically nonsignificant.

View larger version (22K): [in a new window]   Figure 3 Cardiopulmonary exercise testing before and after nonsurgical myocardial reduction. Peak VO2 = peak oxygen consumption; AT = anaerobic threshold. *p < 0.001 compared with baseline. n.s. = statistically nonsignificant.

View larger version (15K): [in a new window]   Figure 4 Functional classes (New York Heart Association [NYHA]) and quality of life scores, determined from a standard questionnaire before and after nonsurgical myocardial reduction. *p < 0.001 compared with baseline. n.s. = statistically nonsignificant.