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Progression to chronic atrial fibrillation after pacing: the Canadian Trial of Physiologic Pacing

Allan C. Skanes, MD^*, Andrew D. Krahn, MD, FACC^*, Raymond Yee, MD, FACC^*, George J. Klein, MD, FACC^*, Stuart J. Connolly, MD, FACC^c, Charles R. Kerr, MD, FACC, Michael Gent, DSc, Kevin E. Thorpe, MMath, Robin S. Roberts, MTech for the CTOPP Investigators

^* Arrhythmia Service, Division of Cardiology, University of Western Ontario, London, Ontario, Canada
Department of Medicine, University of British Columbia, Vancouver, British Columbia, Canada
^c The Department of Medicine, McMaster University, Hamilton, Ontario, Canada
Clinical Epidemiology and Biostatistics, McMaster University, Hamilton, Ontario, Canada

View larger version (18K): [in a new window]   Figure 1 Cumulative risk of the development of chronic atrial fibrillation over time for the groups receiving ventricular (V) and physiologic (P) pacing. The curves diverge between 6 and 12 months and continue to diverge over time.

View larger version (22K): [in a new window]   Figure 2 Distribution of treatment effect of physiologic pacing by subgroup. The hazard ratios (solid circle) and the 95% confidence intervals are plotted for the treatment effect of physiologic pacing. Associated p values are shown. See text for details. Afib = atrial fibrillation; Hypten = hypertension; LVF = left ventricular function; MICAD = myocardial infarction or coronary artery disease; SA = sinoatrial.