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Mild-to-moderate hypertriglyceridemia in young men is associated with endothelial dysfunction and increased plasma concentrations of asymmetric dimethylarginine

Pia Lundman, MD^a , Maria J. Eriksson, MD, PhD, Markus Stühlinger, MD^||, John P. Cooke, MD, PhD^||, Anders Hamsten, MD, PhD^a and Per Tornvall, MD, PhD^a

^a Department of Cardiology, Karolinska Institutet, Stockholm, Sweden
Department of Clinical Physiology, Karolinska Institutet, Stockholm, Sweden
Atherosclerosis Research Unit, King Gustaf V Research Institute, Karolinska Institutet, Stockholm, Sweden
Department of Medicine, Danderyd Hospital, Stockholm, Sweden
^|| Falk Cardiovascular Research Center, Stanford University School of Medicine, Stanford, California, USA

View larger version (12K): [in a new window]   Figure 1 Box plots of relative change in endothelium-dependent flow-mediated vasodilation (FMD) after reactive hyperemia in hypertriglyceridemic (HTG) (n = 15) and control (n = 15) subjects (0.6 ± 0.83% vs. 4.5 ± 2.0%, p < 0.0001). The box plot displays 25th, 50th (median) and 75th percentiles in the box and 5th and 95th percentiles as horizontal lines outside the box. All values above or below these percentiles are plotted as points.

View larger version (15K): [in a new window]   Figure 2 Box plots of common carotid intima-media thickness (IMT) in hypertriglyceridemic (HTG) (n = 15) and control (n = 15) subjects (0.55 ± 0.05 mm vs. 0.53 ± 0.06 mm, NS). The box plot displays 25th, 50th (median) and 75th percentiles in the box and 5th and 95th percentiles as horizontal lines outside the box. All values above or below these percentiles are plotted as points.