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Failure of aldosterone suppression despite angiotensin-converting enzyme (ACE) inhibitor administration in chronic heart failure is associated with ACE DD genotype

Mariantonietta Cicoira, MD^*, Luisa Zanolla, MD^*, Andrea Rossi, MD^*, Giorgio Golia, MD^*, Lorenzo Franceschini, MD^*, Giulio Cabrini, MD, Alberto Bonizzato, BSc, Maristella Graziani, PhD, Stefan D. Anker, MD, PhD ^||, Andrew J. S. Coats, DM, FACC and Piero Zardini, MD^*

^* Divisione di Cardiologia, Università degli Studi di Verona, Verona, Italy
Laboratorio di Patologia Molecolare, Centro Regionale Fibrosi Cistica, Ospedale Civile Maggiore, Verona, Italy
Laboratory of Clinical Chemistry, Ospedale Civile Maggiore, Verona, Italy
Department of Cardiac Medicine, National Heart & Lung Institute, London, United Kingdom
^|| Franz Volhard Klinik (Charite, Campus Berlin-Buch) at Max Delbrück Centrum for Molecular Medicine, Berlin, Germany

View larger version (48K): [in a new window]   Figure 1 Genotype distribution of 132 chronic heart failure patients with and without aldosterone escape despite long-term treatment with angiotensin-converting enzyme inhibitors. Results are reported as percentages of total (graph) and absolute numbers (table). D = deletion; DD = deletion allele; I = insertion; ID = heterozygotes; II = homozygotes.