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Association of angiotensinogen m235t and a(-6)g gene polymorphisms with coronary heart disease with independence of essential hypertension: the procagene study

José C. Rodríguez-Pérez, MD, PhD^*, Francisco Rodríguez-Esparragón, PhD^*, Octavio Hernández-Perera, PhD^*, Aránzazu Anabitarte, RN^*, Antonio Losada, MD^*, Alfonso Medina, MD, PhD^*, Enrique Hernández, MD, PhD^*, Dolores Fiuza, MD^*, Octavio Avalos, MD, PhD^*, Carla Yunis, MD, Carlos M. Ferrario, MD for the PROCAGENE Study Investigators

^* Research Unit, Hemodynamic-Cardiology and Nephrology Services, Hospital de Gran Canaria Dr. Negrín, Las Palmas de Gran Canaria, Canary Islands, Spain
Hypertension and Vascular Disease Center, Wake Forest University School of Medicine, Winston-Salem, North Carolina, USA

View larger version (13K): [in a new window]   Figure 1 Estimates of risk of coronary heart disease associated with polymorphism in the genes coding for angiotensin-converting enzyme (ACE) and angiotensinogen (AGT). (A) Odds ratio (OR) according to genotypes for the ACE (I/D), AGT (M235T) or AGT A(-6)G polymorphisms. (B) Analysis of the risk associated with a determined genotype combination of ACE (I/D) and AGT (M235T) or AGT A(-6)G polymorphisms in relation to the remaining possible combinations. Genotype abbreviations are the same as those shown in Table 2. *p < 0.05.