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Modulation of oxidative stress by a selective inhibition of angiotensin II type 1 receptors in MI rats

^a Institute of Cardiovascular Sciences, St. Boniface General Hospital Research Centre and Department of Physiology, Faculty of Medicine, University of Manitoba, Winnipeg, Canada

View larger version (37K): [in a new window]   Figure 1 Myocardial antioxidant enzyme activities in the 16-week control (CONT) and myocardial infarction (MI) rats with and without losartan treatment. Data are mean ± standard error of the mean, from 5 to 7 animals, with each assay done in duplicate. (A) catalase; (B) glutathione peroxidase; (C) superoxide dismutase. *Significantly different from the respective control group; @Significantly different (p < 0.05) from the respective untreated control and MI groups.

View larger version (41K): [in a new window]   Figure 2 Redox ratio (A) and lipid hydroperoxide content (B) in the 16-week control and MI rats with and without losartan treatment. Data are mean ± SEM from 8 to 10 animals with each assay done in duplicate. *Significantly different (p < 0.05) from the respective control group; @Significantly different from the respective untreated control or myocardial infarction group.