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Recurrent unstable angina after directional coronary atherectomy is related to the extent of initial coronary plaque inflammation

Martijn Meuwissen, MD^*, Jan J. Piek, MD^*,1, Allard C. van der Wal, MD, Steven A. J. Chamuleau, MD^*, Karel T. Koch, MD^*, Peter Teeling, RT, Chris M. van der Loos, PhD, Jan G. P. Tijssen, PhD^* and Anton E. Becker, MD, FACC

^* Department of Cardiology, Academic Medical Center, University of Amsterdam, Amsterdam, The Netherlands
Department of Cardiovascular Pathology, Academic Medical Center, University of Amsterdam, Amsterdam, The Netherlands

View larger version (23K): [in a new window]   Figure 1 Flow chart of the total number of study patients, subcategorized into those with and those without angina within one-year follow-up and further subcategorized into those with recurrent unstable angina and those with recurrent stable angina.

View larger version (15K): [in a new window]   Figure 2 Incidence of recurrent unstable angina pectoris (UAP) within one year after directional coronary atherectomy after separation of the extent of macrophages and T lymphocytes in tertiles, defined by the distribution of the levels of recurrent UAP. Distribution of tertiles is as follows: for macrophages, higher (31%); middle (21% to 31%) and lower (21%); for T lymphocytes, higher (33 cells/mm2), middle (18 to 33 cells/mm2) and lower (18 cells/mm2). *p < 0.0001 for trend (Cochrane Armatege test).