Advertisement






Click here for more guidelines.
CME Topic Collections Past Issues Search Current Issue Home
     

J Am Coll Cardiol, 2000; 36:2204-2211
© 2000 by the American College of Cardiology Foundation
This Article
Right arrow Abstract Freely available
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Krasuski, R. A.
Right arrow Articles by Bashore, T. M.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Krasuski, R. A.
Right arrow Articles by Bashore, T. M.

Inhaled nitric oxide selectively dilates pulmonary vasculature in adult patients with pulmonary hypertension, irrespective of etiology

Richard A. Krasuski, MD*, John J. Warner, MD*, Andrew Wang, MD*, J. Kevin Harrison, MD*, Victor F. Tapson, MD{dagger} and Thomas M. Bashore, MD*

* Division of Cardiology, Department of Medicine, Duke University Medical Center, Durham, North Carolina, USA
{dagger} Division of Pulmonary and Critical Care, Department of Medicine, Duke University Medical Center, Durham, North Carolina, USA



View larger version (17K):

[in a new window]
 
Figure 1 Response to iNO. The majority of the response is seen at 10 ppm of NO in both groups. However, patients with SPH show a trend toward a further pressure reduction with higher doses of iNO.

 




 
  CME Topic Collections Past Issues Search Current Issue Home

Advertisement