Ability of troponin I to predict cardiac events in patients admitted from the emergency department
Michael C. Kontos, MD, FACCa,b,
F. Philip Anderson, PhDc,
Ramin Alimard, MDa,
Joseph P. Ornato, MD, FACCb,
James L. Tatum, MDd and
Robert L. Jesse, MD, PhD, FACCa
a Department of Internal Medicine, Cardiology Division, Medical College of Virginia, Hospitals of Virginia, Commonwealth University, Richmond, Virginia, USA
b Department of Emergency Medicine, Medical College of Virginia, Hospitals of Virginia, Commonwealth University, Richmond, Virginia, USA
c Department of Pathology, Clinical Chemistry Division, Medical College of Virginia, Hospitals of Virginia, Commonwealth University, Richmond, Virginia, USA
d Department of Radiology, Nuclear Medicine Division, Medical College of Virginia, Hospitals of Virginia, Commonwealth University, Richmond, Virginia, USA
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Figure 1 Receiver-operating characteristic curve using MI as the end point. A troponin I value of 1.0 ng/ml was the optimal cutoff value.
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Figure 2 Outcomes in patients with (open bars) and without (solid bars) TnI elevations. Comp = significant complications; D = death; M = myocardial infarction; Sig = significant coronary disease.
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Figure 3 Outcomes in patients with (open bars) and without (solid bars) an ischemic ECG. Comp = significant complications; D = death; M = myocardial infarction; Sig = significant coronary disease.
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Figure 4 Comparison of the sensitivity of positive troponin I (open bars) and the ECG (solid bars) for predicting end points. Comp = significant complications; D = death; M = myocardial infarction; Sig = significant coronary disease.
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