Role of ischemia and infarction in late right ventricular dysfunction after atrial repair of transposition of the great arteries
Teri Millane, MD, MRCP*,
Elizabeth J. Bernard, FRACP ,
Edgar Jaeggi, MD*,
Robert B. Howman-Giles, MD, FRACP, DDU,
Roger F. Uren, FRACP, DDU,
Timothy B. Cartmill, FRACS ,
Richard E. Hawker, FRACP* and
David S. Celermajer, PhD, FRACP* 
* Department of Cardiology, New Children’s Hospital, Sydney, NSW Australia
Department of Nuclear Medicine, New Children’s Hospital, Sydney, NSW Australia
Department of Surgery, New Children’s Hospital, Sydney, NSW Australia
Department of Cardiology, Royal Prince Alfred Hospital, Sydney, NSW Australia
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Figure 1 Summary of perfusion data expressed as (a) myocardial segments affected and (b) as subjects affected. Full details are given in the text.
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Figure 2 (a) Tomographic myocardial perfusion study of the right ventricle at rest and during dipyrdamole stress demonstrating moderate reversible perfusion defects in the anterior wall from the apex to the mid-ventricle (frames A2-5 and B5-8); there is also a fixed perfusion defect in the inferior wall (frames A4-7 and B5-8). The anatomy of the myocardial segments is shown schematically in Figure 1b. (b) Schematic representation demonstrating the orientation of the right ventricle in the perfusion study described in a; the approximate transthoracic echocardiographic plane is indicated.
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Figure 3 The correlation of perfusion abnormalities with reduced wall thickening.
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Figure 4 The deterioration of right ventricular ejection fraction with advancing age.
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