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J Am Coll Cardiol, 2000; 35:1007-1015
© 2000 by the American College of Cardiology Foundation
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Early and intermediate-term complications of self-expanding stents limit its potential application in children with congenital heart disease

Yiu-fai Cheung, MBBS*, Shubhayan Sanatani, BSc, MD{dagger}, Maurice P. Leung, MD, FACC*, Derek G. Human, MA, BM, BCL{dagger}, Adolphus K. T. Chau, MBBS* and J. A. Gordan Culham, MD{dagger}

* Division of Pediatric Cardiology, Grantham Hospital, Department of Pediatrics, University of Hong Kong, Aberdeen, Hong Kong
{dagger} Division of Pediatric Cardiology, British Columbia Children’s Hospital, Vancouver, Canada




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Figure 1 Line plots showing: (A) the increase in diameters of the pulmonary arterial stenosis and (B) decrease in systolic pressure gradients before and immediately after implantation of self-expanding Wallstents. For patients with a functional right ventricle, no significant change of the right ventricular to the systemic systolic pressure ratio could be documented (C). Figure 1, part C on facing page.

 


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Figure 2 Chest roentgenograms showing the initial optimal position and subsequent distal migration of the Wallstents implanted in a hypoplastic left pulmonary artery (A and B). Note the change in position and shape of the proximal end of the stent before and after migration (arrow). In another patient with superior caval venous baffle stenosis, anterioposterior projection of radiography showing initial optimal position of the deployed stent for stenosed baffled limb (C). Note the relative position of the stent in relation to the vertebral border. Chest roentgenogram the next morning showed the stent had migrated, with significant alteration of its alignment and relative position to the vertebral border (D). Echocardiography demonstrated encroachment of the stent on the mitral valve.

 


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Figure 3 Patient with tetralogy of Fallot underwent stenting of right pulmonary artery stenosis. Follow-up catheterization at 8 months demonstrated recurrent stenosis of the right pulmonary artery secondary to neointimal proliferation. The stent (14 mm) remains fully expanded (arrows).

 




 
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