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Antimyosin autoantibodies are associated with deterioration of systolic and diastolic left ventricular function in patients with chronic myocarditis

Bernward Lauer, MD^*, Mira Schannwell, MD, Uwe Kühl, MD, Bodo-Eckhard Strauer, MD and Heinz-Peter Schultheiss, MD

^* Klinik für Innere Medizin/Kardiologie, Universität Leipzig-Herzzentrum, Leipzig, Germany
Medizinische Klinik und Poliklinik B, Heinrich-Heine-Universität, Düsseldorf, Germany
Abteilung für Kardiologie, Pneumologie und Angiologie, Universitätsklinikum Benjamin Franklin, Berlin, Germany

View larger version (134K): [in a new window]   Figure 1 Representative histologic and immunohistologic pictures of biopsy specimen from patients with myocarditis. A: histology (borderline myocarditis). B: immunohistology (lymphocytic myocarditis).

View larger version (12K): [in a new window]   Figure 2 Histological and immunohistological analysis of the endomyocardial biopsies in 33 patients with chronic myocarditis at baseline and at follow-up (six months). Acute MC = acute myocarditis (histology); bord MC = borderline myocarditis (histology); lymph MC = lymphocytic myocarditis (immunohistology); no MC = no myocarditis (histology or immunohistology).

View larger version (11K): [in a new window]   Figure 3 Antimyosin autoantibodies in patients with chronic myocarditis at baseline and at follow-up (six months). AMAAB = antimyosin autoantibodies.