Low-level exercise echocardiography detects contractile reserve and predicts reversible dysfunction after acute myocardial infarction
Comparison with low-dose dobutamine echocardiography
Etienne P. Hoffer, MDa,
Walthère Dewé, MSca,
Carmine Celentano, RNa and
Luc A. Piérard, MDa
a Department of Cardiology, University Hospital Sart Tilman, Liège, Belgium
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Figure 1 End-diastolic (ED) and end-systolic (ES) stop-frame images of illustrative echocardiograms (apical two-chamber views) obtained in a patient showing inferior akinesia at rest (arrows), mild hypokinesis during low-level exercise echocardiography (LLEE), normal thickening during low-dose dobutamine echocardiography (LDDE) and at follow up (FU).
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Figure 2 Evolution of the baseline dyssynergic segments with low-level exercise echocardiography (LLEE), low-dose dobutamine echocardiography (LDDE) and at follow-up study (FU).
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Figure 3 Conventional M-mode echocardiogram recordings obtained at rest, during low-level exercise echocardiography (LLEE), low-dose dobutamine echocardiography (LDDE) and at follow-up (FU) in a patient with inferior myocardial infarction. Images were acquired in parasternal short axis view. End-diastolic values were recorded at the beginning of QRS complex and in end-systolic values on top of T-wave. Absolute systolic thickening was 0.1 mm at baseline, 2.9 mm during LLEE 4.8 mm during LDDE, and 2.4 mm at baseline on the follow-up echocardiogram.
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Figure 4 Relation between absolute systolic thickening in the center of the dysfunctional area measured during low-level exercise echocardiography (LLEE) and low-dose dobutamine echocardiography (LDDE), and at follow-up (FU) in the whole-study patients (n = 45).
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