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Neuroendocrine activation in heart failure is modified by endurance exercise training

Randy W. Braith, PhD^*, Michael A. Welsch, PhD, Matthew S. Feigenbaum, PhD, Heidi A. Kluess, MS and Carl J. Pepine, MD, FACC^*

^* Center for Exercise Science, College of Health and Human Performance and the College of Medicine, University of Florida, Gainesville, Florida, USA
Department of Kinesiology, Louisiana State University, Baton Rouge, Louisiana, USA
Department of Health and Exercise Science, Furman University, Greenville, South Carolina, USA

View larger version (44K): [in a new window]   Figure 1 Relative changes in angiotensin II (ANG II), aldosterone (ALDO), arginine vasopressin (AVP), and atrial natriuretic peptide (ANP) after four months of endurance exercise training or control period. Data are mean ± SEM. *p 0.05 After training versus before training.

View larger version (39K): [in a new window]   Figure 2 Angiotensin II (top panel) and aldosterone (bottom panel) concentrations drawn at rest in the standing posture for age-matched healthy control subjects; heart transplant recipients; heart failure patients before four months’ exercise training; and heart failure patients after four months’ exercise training. Data are mean ± SEM. *p 0.05 heart failure before training versus the other three groups. p 0.05 versus heart failure after training and normal control group.

View larger version (40K): [in a new window]   Figure 3 Arginine vasopressin (top panel) and atrial natriuretic peptide (ANP) (bottom panel) concentrations drawn at rest in the standing posture for age-matched healthy control subjects; heart transplant recipients; heart failure patients before four months’ exercise training; and heart failure patients after four months’ exercise training. Data are mean ± SEM. *p 0.05 heart failure before training versus the other three groups. p 0.05 versus heart failure after training and normal control group.