The effects of biphasic and conventional monophasic defibrillation on postresuscitation myocardial function
Wanchun Tang, MD*  ,
Max Harry Weil, MD, PhD, FACC* ,
Shijie Sun, MD* ,
Hitoshi Yamaguchi, MD*,
Heitor P. Povoas, MD*,
Andreja Marn Pernat, MD* and
Joe Bisera, MSEE*
* The Institute of Critical Care Medicine, Palm Springs, California, USA
The University of Southern California School of Medicine, Los Angeles, California, USA
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Figure 1 Effects of biphasic or monophasic defibrillation waveform on heart rate and mean aortic pressure (MAP) after either 4 or 7 min of untreated VF. Values are mean ± SD. BL = baseline. *p < 0.05 vs. biphasic, **p < 0.01 vs. biphasic.
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Figure 2 Effects of biphasic or monophasic defibrillation waveform on postresuscitation myocardial function after 4 min of untreated VF. Values are mean ± SD. BL = baseline, CO = cardiac output, SV = stroke volume, LPWS = systolic left ventricular posterior wall thickness, LPWD = diastolic left ventricular posterior wall thickness. *p < 0.05 vs. biphasic, **p < 0.01 vs. biphasic.
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Figure 3 Effects of biphasic or monophasic defibrillation waveform on postresuscitation myocardial function after 7 min of untreated VF. Values are mean ± SD. BL = baseline, CO = cardiac output, SV = stroke volume, LPWS = systolic left ventricular posterior wall thickness, LPWD = diastolic left ventricular posterior wall thickness. *p < 0.05 vs. biphasic, **p < 0.01 vs. biphasic.
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