Ability of troponins to predict adverse outcomes in patients with renal insufficiency and suspected acute coronary syndromes: a case-matched study
Frederick Van Lente, PhDa,
Ellen S. McErlean, MSNa,
Sue A. DeLuca, BSNa,
W. Franklin Peacock, MDa,
J. Sunil Rao, PhDa and
Steven E. Nissen, MD, FACCa
a Department of Clinical Pathology, The Department of Cardiology, The Division of Nursing, and The Department of Emergency Medicine, The Cleveland Clinic Foundation, Cleveland, Ohio, USA
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Figure 1 Receiver operating characteristic curves for (A) cardiac troponin T (cTnT) and (B) cardiac troponin I (cTnI) in predicting initial hospitalization adverse events in patients with (dotted line) and without (solid line) renal insufficiency. The lines represent prognostic sensitivity and false positive rates at increasing threshold values. The AUC and standard error are shown for each curve and the upper-left inflection points are indicated by an arrow and the corresponding sensitivity and specificity are shown in Table 3.
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Figure 2 Receiver operating characteristic curves for predicting adverse outcomes by 6 months for (A) cardiac troponin T (cTnT), (B) cardiac troponin I (cTnI) and (C) creatine kinase MB (CKMB). The dotted line represents the renal group and the solid line the nonrenal group. The lines represent prognostic sensitivity and false positive rates at increasing threshold values. The AUC and standard error are shown for each curve and the upper-left inflection points are indicated by an arrow and the corresponding sensitivity and specificity are shown in Table 3.
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