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Impairment of the nitric oxide–mediated vasodilator response to mental stress in hypertensive but not in hypercholesterolemic patients

^a Cardiology Branch, National Heart, Lung and Blood Institute, National Institutes of Health, Bethesda, Maryland, USA

View larger version (24K): [in a new window]   Figure 1 Bars show the percent changes in forearm blood flow from baseline in response to mental stress in normotensive, hypertensive and hypercholesterolemic subjects. Values represent mean ± SEM.

View larger version (20K): [in a new window]   Figure 2 Bars show the effect of nitric oxide synthesis inhibition by NG-monomethyl-L-arginine (L-NMMA, 4 µmol/min) on the percent changes in forearm blood flow from baseline in normotensive, hypertensive and hypercholesterolemic subjects. Values represent mean ± SEM. Open box = saline; solid box = L-NMMA.

View larger version (21K): [in a new window]   Figure 3 Bars show the percent changes in forearm blood flow from baseline in response to intraarterial infusion of acetylcholine (30 µg/min) and sodium nitroprusside (3.2 µg/min) in normotensive (NV), hypertensive (HT) and hypercholesterolemic (HC) subjects. Values represent mean ± SEM.