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J Am Coll Cardiol, 1998; 32:373-379
© 1998 by the American College of Cardiology Foundation
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Production of oxidative products of nitric oxide in infarcted human heart

Kazuhide Akiyama, MDa, Akio Kimura, MDa, Hiroshi Suzuki, MD*, Youichi Takeyama, MD*, Tracy L. Gluckman, BSa, Artin Terhakopian, BSa, Takashi Katagiri, MD*, Ki-Young Suh, BSa, James Roseto, BSa and Richard J. Bing, MD, FACCa

a Department of Experimental Cardiology, Huntington Medical Research Institutes, Pasadena, California, USA
* Third Department of Internal Medicine, Showa University, School of Medicine, Tokyo, Japan



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  		Figure 1 Mean plasma concentrations (µmol/L) of NOx from arterial (closed circles), peripheral venous (open triangles) and coronary sinus (open squares) plasma in control patients and in patients with AMI. Plasma NOx concentrations are significantly elevated above control values, at 48 and 72 h after onset of symptoms (p < 0.01). Values for NOx in coronary sinus plasma significantly exceed those in peripheral venous plasma from 24 to 72 h after onset of symptoms (p < 0.05). The NOx values in coronary sinus plasma also significantly exceed those in arterial plasma from 24 to 72 h after onset of symptoms (p < 0.05).

 


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  		Figure 2 NOx concentrations in arterial plasma of individual AMI patients increased from 24 to 48 h after onset of symptoms. The peak NOx concentration usually occurred from 48 to 72 h after onset of symptoms.

 




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Copyright © 1998 by the American College of Cardiology Foundation.