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Figure 4 Activity of protein kinase C (PKC). Myocardial PKC activity in the particulate fraction was increased in noninfarcted viable myocardium of the untreated myocardial infarction (MI) animals (MI/control [C]) compared with shams. Treatment of MI animals with candesartan (MI/angiotensin II type 1 receptor blocker [ARB]) reduced PKC activity in the particulate fraction, compared with MI/C. *p < 0.05 MI/C vs. sham; {dagger}p < 0.05 MI/ARB vs. MI/C; n = 5 in each group.