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Figure 5 Representative original traces showing recovery from intracellular acidosis after stepwise washout of 30 mM NH₄Cl of endocardial tissue preparations from infarcted rat hearts. Effects of chronic treatment with the angiotensin-converting enzyme inhibitor ramipril, the AT₁ receptor antagonist valsartan and the AT₂ receptor antagonist PD 123319 on puffering power (ß_i) and proton efflux (J_H) seven days after myocardial infarction (MI) compared with sham-operated and placebo-treated animals with MI. ami = amiloride; PSS = physiological saline solution.